Diagnosis and treatment of diabetic foot infections

Executive summary

1. foot diseases in patients with diabetes, disease, and health care professionals to amputation of the extremity and low cause trips can be self-destructive.

2. diabetic foot by local (feet) into the blood stream and systemic (metabolic) issues and the associated management, preferably moltadaskaplanari fotcari team (A-II) attention is required by. These diseases should be added to the team to manage infectious diseases expert, or a medical or auto access, a microbioligist (B-II) is.

3. major pradaspasang iolsraon adjoining in these diseases, which is usually the element for foot neuropathy is related to. Peripheral vascular disease and various ammnalogakal ankle a secondary role. 4. kokaka arubak Gram-positive infections (especially aoreos area) are predominant pathogens in diabetic foot. That chronic wounds or who recently have received antibiotic therapy and negative rods also atents g with askhamia foot or gangrene can aoblagty anaerobic pathogens.

5. wound infection in clinical signs and symptoms of local (and sometimes sistemitic) must be assessed on the basis of inflammation. Except in cases of infection, laboratory investigations (including major) aostumilatas diagnasang are restricted for use (B-II) ۔

6. specamance infection, except perhaps that in all these cases, experimental light and treatment before antibiotic treatment to properly send obtained before starting culture (B-III). Baopse, ulcer surgery treatment method or wish to wound tissue obtained by specamance broom specamance are preferable (A-I).

7. diagnosis of soft-tissue imaging study or better deep, prolent can help define a set and usually need bone results in detection. Simple radevgrapei in many cases may be suitable, but MRI (preferred isotope scanning), more sensitive and specific, particularly for soft-tissue research.

8. clinical and laboratory features easily by infection assisabali (B II) should be based on the severity of their classification. Absorbed into the blood stream and systemic toxicity artral specific tissue involved, or the presence of metabolic instability of prfuon adequacy of these are most important. Category classification of patient and limb and, therefore, the risk for the management of the venue immediately and helps determine the status of.

9. the evidence available antibiotic therapy (D-III), as well as treating clinically ananfactid does not support had an ulcer.
Antibiotic therapy is required for practically all the infected wound, but it often is insufficient without proper wound care.

10. an experimental antibiotic regimen of potential infections and ataalogak no-cost (s) (B-II) on the basis of the severity of select. Arubak kokaka only purpose on Gram-positive therapy have recently been found in patients with antibiotic therapy (A-II)-moderate infections may be enough for. Brodspctorm experimental therapy need not be normal but severe infections with antibiotics pending results of the culture, and what is the point for Susceptibility data (B-III). There is no current antibiotic therapy and antibiotic susceptibility of local data, especially the spread of resistant s. aoreos boss (Mirza) or other resistant organisms. The results of both the culture and the capability of data and experimental definitive therapy regimen (C-III) should be based on clinical response.   more >>